Dihydroartemisinin up‐regulates VE‐cadherin expression in human renal glomerular endothelial cells

نویسندگان

  • Liqun Li
  • Xiaocui Chen
  • Fengyun Dong
  • Qiang Liu
  • Caiqing Zhang
  • Dongmei Xu
  • Thaddeus D Allen
  • Ju Liu
چکیده

The antimalarial agent dihydroartemisinin (DHA) has been shown to be anti-inflammatory. In this study, we found that DHA increased the expression of the junctional protein vascular endothelial (VE)-cadherin in human renal glomerular endothelial cells. In addition, DHA inhibited TGF-β RI-Smad2/3 signalling and its downstream effectors SNAIL and SLUG, which repress VE-cadherin gene transcription. Correspondingly, DHA decreased the binding of SNAIL and SLUG to the VE-cadherin promoter. Together, our results suggest an effect of DHA in regulating glomerular permeability by elevation of VE-cadherin expression.

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عنوان ژورنال:

دوره 22  شماره 

صفحات  -

تاریخ انتشار 2018